The two keys to avoiding gout recurrence are to know how to suffer less, and to watch for relatives and friends

Why do gout attacks occur more frequently?

The main reason for gout attacks is that high uric acid leads to the formation of urate crystals in the joints, which stimulate inflammation, redness, swelling, heat, and pain in the joints.

When gout is first detected, there are fewer urate crystals. If you do not regulate uric acid lowering at this time, more and more uric acid salts will deposit in the joints, and eventually the attacks will become more frequent.

Some studies have shown that blood uric acid<300 μ At mol/L, the recurrence rate of gout was less than 10%; And blood uric acid>540 μ At mol/L, the recurrence rate of gout is 80%, which is<300 μ Mol/L of patients.

Therefore, the root cause of the increasing frequency of gout attacks is that uric acid is not reduced.

Why do you have more frequent seizures when taking medication?

As mentioned earlier, urate deposits in joints can cause joint pain. Similarly, when existing urate crystals dissolve, they can also stimulate joints and cause gout attacks.

For some patients who have just started taking drugs to reduce uric acid, lowering uric acid can cause the dissolution of uric acid crystals, forming "microcrystals," which can stimulate joints.

At this time, frequent episodes of gout can be seen as a sign of effective uric acid lowering or dissolution of uric acid crystals.

Two keys to avoiding recurrence of gout

　　1

In the short term, keep your mouth shut

This is easy for everyone to understand.

For example, it is now the season to eat crayfish. If you eat midnight snack, beer crayfish, and some barbecue at night, uric acid will rise sharply, and then gout will occur.

　　2

Standardize treatment to keep uric acid up to standard

The key to reducing repeated episodes of gout is to continuously meet the standard of blood uric acid. Patients without gout stones should reduce their blood uric acid value to 360 μ Below mol/L. Patients with gout stones should reduce their blood uric acid to 300 μ Below mol/L.

However, during uric acid lowering therapy, especially at the beginning of uric acid lowering, due to the fact that uric acid lowering is particularly prone to induce gout, two points need to be noted at this time:

Try to reduce uric acid steadily

Due to large individual differences, the rate of lowering uric acid with the same dose of uric acid lowering drugs often varies among different individuals. In particular, benzbromarone and fepristone have strong uric acid lowering effects and are prone to disease fluctuations. Therefore, the pursuit of rapid lowering of uric acid cannot be blindly pursued.

2. Applying Seizure Prevention Drugs

There are often gout patients who stop taking a box of uric acid lowering drugs because they feel that "this drug is useless, it's okay not to take it, gout occurs more frequently after taking it.".

Therefore, when starting uric acid lowering therapy, it is particularly important to use drugs such as colchicine to prevent gout attacks. This can not only alleviate short-term recurrent pain, but also strengthen everyone's determination to adhere to reducing uric acid, thereby achieving the long-term goal of gout treatment.