Post Date December, 03 2023
Prostate atrophy, for example, is like a wilting eggplant that has lost water. Of course, the atrophy of the prostate is not due to dehydration, but rather to a decrease in prostate metabolism and nutritional supply function caused by stiffness and sclerosis (also known as "fibrosis").
It is understood that a large number of capillaries are distributed inside the prostate gland. These capillaries, through normal blood circulation, are responsible for supplying nutrients to the prostate and promoting prostate metabolism.
When the current prostate gland becomes stiff and sclerotic, it will directly cause damage to the internal capillaries of the prostate, thereby affecting the normal blood circulation of the prostate and hindering its normal metabolism and nutrient intake function. When the prostate gland cannot receive nutrients normally, it is prone to atrophy.
This is like an eggplant still hanging on a branch, suddenly being cut off from its roots, which it relies on to absorb nutrients. The nutrients contained in the soil cannot be continuously transmitted to eggplants through the roots, leading to dehydration and wilting.
To get to the point, let's take a look at how prostate atrophy affects male urination function
Firstly, prostate atrophy is often accompanied by a decrease in prostate elasticity.
As is well known, the prostate has a continuous pipeline, which is medically referred to as the "posterior urethra of the prostate". This pipeline is actually a part of the male urethra. The reason why the prostate can control male urination is fundamentally through its own contraction and relaxation, achieving compression and release of this pipeline.
As mentioned earlier, prostate atrophy occurs under the premise of prostate stiffness and sclerosis. Prostate stiffness and sclerosis is actually a process in which the texture of prostate gland tissue becomes harder and its elasticity decreases.
The texture of the prostate becomes harder and its elasticity decreases, making it unable to achieve normal contraction and relaxation, which in turn affects the compression and release of the posterior urethra of the prostate and ultimately affects the normal urination function of men.
Secondly, prostate atrophy involves a process of inward contraction.
It is understood that the prostate gland is surrounded by a flexible and highly elastic capsule. This envelope, like a balloon, has very good elasticity. Blow air on it, it will become larger; If you deflate it, it will shrink. This is precisely the reason why the prostate can exhibit both hyperplasia and atrophy.
The atrophy of the prostate gland is like deflating an inflated balloon. During this process of "deflation", the prostate capsule will continuously contract inward as the volume of the outer layer of the prostate tissue shrinks, tightly gripping the inner layer of the prostate tissue, forcing the prostate to continuously contract inward, thereby compressing the posterior urethra of the prostate, ultimately affecting the normal urination function of men.
Furthermore, prostate atrophy is often accompanied by damage to neural tissue.
Based on the content mentioned earlier, in summary, the occurrence of prostate atrophy roughly goes through the following processes: stiffness and sclerosis → damage to capillaries → decreased metabolism and nutrient supply → prostate atrophy.
Experts point out that since stiffness and sclerosis can cause damage to the capillaries of the prostate, the prostate nerve tissue, which is more fragile than the capillaries, naturally cannot escape fate.
It is understood that the contraction and relaxation of the prostate depend to a large extent on the control and regulation of the neural tissue inside the prostate. In other words, once these neural tissues are damaged, they will directly affect the normal contraction and relaxation of the prostate, leading to a decrease in male urination function.
