Postpartum women also undergo four measures for pre syndrome, which should be remembered

Premenstrual syndrome is a disease that we should pay attention to, and its causes are also multifaceted, especially in postpartum women who should pay attention to active prevention and treatment methods after getting sick.

Causes of postpartum premenstrual syndrome

1. Fluid retention: Systemic fluid retention caused by excessive aldosterone hormone is often used to explain the formation of premenstrual syndrome. It is known that progesterone can block the effect of aldosterone on the renal tubules, which is beneficial for the excretion of urine sodium. However, progesterone causes sodium loss, leading to a compensatory increase in aldosterone. In addition, the activity of progesterone in the second half of menstruation to convert into salt corticosteroids such as deoxycortisone is enhanced, leading to an increase in aldosterone secretion before menstruation, which reaches its peak before menstruation. Therefore, the increase in aldosterone excretion during the luteal phase is physiological, in order to maintain stable plasma sodium levels, Moreover, there was no significant difference in the determination of plasma aldosterone levels in patients with premenstrual syndrome compared to the control group, which does not support the above theory.

2. Psychological stimulation: Due to the widespread and disconnected nature of symptoms in the early stages of the syndrome, using a placebo or receiving psychological and psychological treatment can also be effective. Many scholars have proposed that the reason for mental and social factors causing physical and mental dysfunction is Parker's synthesis of many scholars' opinions. Personality and environmental factors are extremely important for the occurrence of early syndrome symptoms, and the appearance of symptoms reflects unresolved conflicts in the patient's heart. Tracing the patient's life history often reveals significant psychological stimuli, such as childhood misfortunes and trauma, parental and family discord, poor academic performance, and breakdowns, which may be important factors in emotional changes before the event.

3. Prolactin: Increased excretion of prolactin (PRL): In recent years, there has been a significant debate surrounding the issue of increased PRL excretion as an important etiological factor for PMS. The plasma PRL concentration has a circadian rhythm, with the highest level during sleep, and there are significant changes between each person's days. The PRL level reaches its peak during ovulation, and the average PRL level during luteal phase is higher than that during follicular phase. Some early syndrome patients have higher average blood PRL concentrations than healthy women throughout the entire menstrual cycle, especially in the early stages. Treatment with bromocriptine inhibits PRL secretion and significantly alleviates symptoms. The increase in PRL levels during the luteal phase can be accompanied by a decrease in progesterone excretion or a decrease in FSH/LH levels, which theoretically supports the association between increased PRL levels and the formation of premenstrual syndrome in some aspects. However, there was no significant difference in PRL levels observed between normal and pre syndrome patients. The regulatory effect of PRL on osmotic pressure is significant in animals and has little impact on humans. It only affects the breast, and the balance of local osmotic pressure may expand the breast and cause pain. In addition, women with high PRL rarely experience symptoms of pre syndrome. The application of bromocriptine therapy only alleviates breast symptoms, while other symptoms have no significant effect. There is a lack of reliable and strong evidence for increasing PRL emissions.