Post Date December, 02 2023
The skin near the head of the penis folds inward into a double layer, covering the penis, known as the foreskin.
There is slight adhesion between the foreskin and the head of the penis in newborns and infants, which gradually disappears around the age of 1 year. The foreskin and head of the penis naturally separate, so the foreskin in children appears longer, which is not an anomaly. If, during adolescence, the glans penis still refuses to reveal itself, or if in childhood, the glans penis is tightly wrapped in a foreskin, then attention should be paid.
Both phimosis and redundant prepuce are born with dysplasia.
Phimosis refers to the condition where the opening of the foreskin is too small and the foreskin cannot be turned up to expose the glans penis; If the foreskin is too long, it covers all of the glans penis, and the opening of the foreskin is not small, which can barely be turned up to expose some of the glans penis. If there is phimosis and foreskin too long, and the glans penis cannot be exposed, then this "harm trilogy" will sound.
Disgusting smegma
Just as various parts of the skin secrete sebum, the sebaceous glands of the foreskin also secrete sebum. Due to phimosis or excessive foreskin, the foreskin cannot be lifted upwards, resulting in the accumulation of sebum in the gap between the inner surface of the foreskin and the glans penis. At the same time, urine will also seep into this gap and react with the sebum, becoming smegma and producing strange odor. The smegma accumulated for a long time will become a hard lump, which makes people mistakenly think that it is a "tumor".
Smegma is a chemical carcinogen. Many experiments have confirmed that it has a strong carcinogenic effect. For example, when horse smegma is inoculated into mice, it can cause skin malignant tumors. Inoculating mice with human smegma can also induce cervical cancer in female mice. Modern times have confirmed that the specific hazards of smegma are as follows:
Directly stimulating the head of the penis can induce penile cancer.
- Causes frequent inflammation of the foreskin and penis head, and long-term inflammatory stimulation will cause degeneration and deterioration of local histiocyte of the penis.
Creating a harsh environment at the head of the penis can trigger precancerous changes in many penile cancers, such as penile angle, penile papilloma, penile leukoplakia, proliferative penile erythema, etc. These types of lesions can easily transform into penile cancer.
Malignant penile cancer
Under the "prelude" of phimosis and redundant prepuce, behind the "theme" of smegma, there will be a "variation" of penis cancer, which is a terrible "climax", because penis cancer is a disease that will take away life.
One day, especially at the age of 50-60, there will be an inexplicable lump on the head of the penis, which is neither painful nor itchy, but it will continue to grow and grow like a camellia, protruding from the surface of the penis and changing its shape. The surface of the lump can also ulcerate, with a foul odor and many secretions. As the condition progresses, nearby lymph nodes expand, and eventually cancer cells metastasize to the liver and lungs, posing a threat to life. Of course, surgery and other methods can be used to cure early penile cancer, but wouldn't it be better if penile cancer could not occur?
Since this "dangerous chain" begins with phimosis and foreskin elongation, it is necessary to take precautions and cut off this "prelude". A "trilogy of harm" cannot be played. The Jewish people perform circumcision on the 8th day after the birth of a baby, so there is almost no occurrence of penile cancer; Muslim Muslims also undergo circumcision in their children between the ages of 4 and 10, which significantly reduces the incidence of penile cancer. Don't these examples fully illustrate the problem?
